Essay Alzheimer Disease

Alzheimer's disease

Table of Content

Introduction ………………………………………………………………………. P.3

Definition…………………………………………………………………………..P.4

Essay Research …………………………………………………………………P.5-7

Conclusion……………………………………………………………………... P.8

References…………………………………………………………………….. P.9

Appendix ……………………………………………………………………..P.10-16

Introduction

The disease is a progressive disease which means that the disease gets worse and over time more functions are lost. The disease first attacks the memory center of the brain, causing people will forget things. This disease is common in people 65 years or older. The causes and symptoms of Alzheimer's are different than other forms of dementia.

Definition

1. oligodendrocyte-cells are smaller than with few prologaciones astrocytes.

1. nucleation-is the beginning of a change of state in the region small but stable.

3.placebo-is a simulated or otherwise medically ineffectual treatment for a disease or other medical condition to deceive the intended recipient.

4. memory-was a term memory for aspects in western classical involving rtetoric.

5. cell- is morphological and functional unit of all being alive.

Essay Research

Alzheimer's disease is a degenerative brain disease for which no cure exists. This disease slowly attacks nerve cells in all parts of the cortex of the brain and some surrounding structures, thereby deteriorating the individual capabilities of emotions, recognize errors, coordinate movement, and remember. When this advanced disease the person loses all memory and mental functioning. Nearly half of all people who are in hospitals and elderly over 85 suffer from Alzheimer's disease. This disease is the fourth leading cause of death in adults and, unless methods are developed for the prevention and treatment of Alzheimer's disease this will be even more common in the coming years. Early symptoms of Alzheimer's disease may be overlooked because they resemble signs of natural aging. These symptoms include forgetfulness, loss of concentration, unexplained weight loss and motor problems, including mild difficulty walking. In healthy individuals, similar symptoms can result from fatigue, grief or depression, illness, loss of vision or hearing, use of alcohol or certain medications, or simply the burden of having to remember too many details immediately. But when memory loss worsens, the family and friends feel that there are serious problems.

Biological factors in the brain for this disease are found twisted nerve cell fibers called tangles neurofibrilatories and sticky protein called beta amyloid. The entangled fibers are damaged microtubules, this is the support structure that permits the flow of nutrients through the neurons. In the protein known as tau is found in these tangles, and some experts believe that this defective version attracts and maintains normal tau proteins commonly help the formation of a healthy microtubule structure. The second test is a high concentration of sticky protein called beta amyloid, which forms patches called neuritic plaques. These plates are outside nerve cells surrounded by the remains of dying neurons. Beta amyloid protein itself is a larger protein called amyloid beta amyloid. And also associated with reduced levels of the neurotransmitter acetylcholine. Neurotransmitters are chemical messengers in the brain. Acetylcholine is part of the cholinergic system, essential for the processes of memory and learning, which is progressively destroyed in patients with Alzheimer's disease. Researchers have identified important proteins, including associated binding protein of retílo reticulum and AMY plates, which are in areas of the brain affected by Alzheimer's disease. ERAB endoplasmic reticulum appears that combines with amyloid beta for attracting new beta amyloid out of the cells. It also appears that high amounts of ERAB enhance the destructive power of its partner protein nerves. AMY plaques resemble both the beta amyloid, which could be detected only through highly sophisticated techniques.

A recent study is that the risk factors for Alzheimer's disease may be different in women and men. Men have a higher risk of developing Alzheimer's disease. Estrogen therapy may help protect older women. Creaneoencefalico trauma to the head can accelerate the development of Alzheimer's disease in individuals who are already susceptible to disease. Some studies have found a link between atherosclerosis, or hardening of the arteries, related to Alzheimer's disease in specific people who carry the gene ApoE4.Hay studies showing that estrogen therapy protects against Alzheimer's disease and impaired mental functioning normally associated with aging. Several studies have reported that women who are making the hormone replacement therapy and have better results in learning and memory test that women are not doing. Found that women who were making the hormone replacement therapy had a reduced risk of Alzheimer's disease in a study, the risk was lower by 60%. Estrogen therapy can help women with Alzheimer's disease. One study found that women with Alzheimer's disease showed mild to moderate mental functioning better after taking estrogen and another reported less evidence of Alzheimer's disease in the brains of women taking estrogen than in those who were not taking . These reports support the animal studies suggest that estrogen triggers a process that leads to the production of acetylcholine, a chemical which is at very low levels in patients with Alzheimer's disease. It also appears that estrogen therapy stimulates blood flow in the brain.

Nonsteroidal antiinflammatory drugs common, such as aspirin, ibuprofen (Advil, Motrin) and naprosyn, have properties that block specific factors in the inflammatory response is believed to play an important role in the degeneration of neurons. A long-term study found that people who took ibuprofen for two years or more had a 50% reduction in the incidence of Alzheimer's disease compared with those not taking the drug. In the same study, the long-term use of aspirin did not appear to confer any benefit, perhaps because the dose was often very low .. Acetaminophen (Tylenol) is not an anti-inflammatory and has no effect on the disease.

Eating habits have to do with a diet low in fat may reduce the risk of Alzheimer's disease. In countries with low-fat diets as in China and Nigeria, the risk of developing Alzheimer's disease is 1% at age 65 years compared with 5% in the United States. To support this, a study in the Netherlands found that diets high in total fat, saturated fat and cholesterol were associated with dementia. Although you should avoid saturated fats and acids transadipicos, some fats, such as adipic acid omega-3, found in fish such as salmon, halibut, swordfish and tuna, are essential for nervous system development and these adipic acid may also help protect against mental decline in old age. Some reports have indicated that the antioxidant properties found in substances such as vitamin C, E and selenium may be protective against mental decline, but evidence is needed to confirm this. Vitamin E is of particular interest. Most foods are rich in this vitamin, but is found in vegetable oils (particularly wheat germ oil), sweet potatoes, avocados, peanuts, sunflower seeds and soybeans

Several studies have reported an increased risk of Alzheimer's disease among people with less education and a lower risk of dementia and Alzheimer's disease in people who remain mentally active. A few experts speculate that learning itself stimulates further growth of neurons and thus can create a larger reserve in the brain so that it takes more time that brain cells are destroy. Others believe that such socio-economic

...